Spinal fracture
Overview
A spinal fracture is a break in one or more vertebrae - ranging from osteoporotic compression fractures to unstable fracture-dislocations. The key clinical question is always: is the spinal cord or nerve roots at risk?
Presentation
•Localised spinal pain - midline tenderness on palpation
•Limb weakness, sensory loss/paraesthesia - indicates cord or root involvement
•Bladder/bowel dysfunction - red flag for cord or cauda equina compromise
•Saddle anaesthesia (perianal sensory loss) - pathognomonic of cauda equina syndrome
•Neurogenic shock - bradycardia, hypotension, warm dry peripheries, priapism; due to loss of sympathetic outflow in high thoracic/cervical injury
UMN vs LMN signs in spinal injury
| Feature | UMN (cord, above L1) | LMN (cauda equina, at/below L1) |
|---|---|---|
| Tone | Spasticity | Flaccidity |
| Reflexes | Hyper-reflexia, clonus | Hyporeflexia |
| Plantars | Upgoing (Babinski +ve) | Downgoing |
| Wasting | Absent early | Present |
Investigations
🥇 First-line
•Plain X-ray (AP and lateral) - initial screening for thoracic/lumbar injuries; may miss posterior element fractures
•First-line (cervical/high suspicion): CT spine - preferred for suspected cervical fracture and any thoracolumbar injury with abnormal X-ray or high clinical suspicion; superior bony detail
🏆 Gold standard
•MRI whole spine - mandatory when neurological deficit present, suspected cord/cauda equina compromise, or no bony injury on CT; evaluates cord, discs, ligaments, and epidural haematoma
Management
All suspected spinal fractures
- 1Immobilise spine - cervical collar and spinal board pre-hospital; log-roll technique
- 2ABCDEs - treat neurogenic shock with IV fluids and vasopressors if bradycardia + hypotension
- 3CT/MRI to define injury and stability
- 4Bloods: FBC, U&E, CRP, bone profile
Neurologically intact / stable fracture
Conservative: analgesia, immobilisation/bracing, physiotherapy; treat underlying osteoporosis (alendronic acid + calcium/vitamin D); consider vertebroplasty/kyphoplasty if VCF pain persists >6 weeks
Neurological deficit / unstable fracture
Urgent surgical decompression and stabilisation; emergency surgery for epidural haematoma or acute neurological deterioration; transfer to regional spinal cord injuries centre
Complications
•Cauda equina syndrome - surgical emergency; bladder/bowel dysfunction and saddle anaesthesia
•Autonomic dysreflexia - injury at/above T6; triggered by noxious stimuli below lesion; severe hypertension, headache, sweating above lesion, bradycardia; can cause seizures and stroke
•Respiratory failure - high cervical injury (above C4) may require ventilatory support
•VTE - DVT prophylaxis with LMWH required due to immobility
•Progressive kyphosis and height loss - complication of multiple osteoporotic VCFs
Prognosis
•Ambulant at presentation → ~80% remain ambulant after treatment
•Complete cord injury (absent motor and sensory function below level) → poor prognosis for neurological recovery
•Incomplete cord injury → significant improvement possible
•Osteoporotic VCFs → increased risk of future fractures and independently associated with increased mortality (respiratory complications from kyphosis); treat underlying osteoporosis
Key Anatomy
•Spinal cord ends at ~L1 (conus medullaris); below L1, nerve roots form the cauda equina
•Injury above L1 → cord damage → upper motor neurone (UMN) signs
•Injury at/below L1 → cauda equina → lower motor neurone (LMN) signs
•Denis three-column model - disruption of ≥2 columns = mechanical instability
Fracture Types
Key fracture patterns
| Fracture | Mechanism | Key feature |
|---|---|---|
| Burst fracture | High-energy axial load (diving, fall from height) | Bone driven into spinal canal - unstable |
| Chance fracture | Flexion-distraction (lap-belt injury) | Through posterior elements |
| Vertebral compression fracture (VCF) | Osteoporosis; trivial/no trauma | Wedge-shaped; anterior body height loss |
| Ankylosing spondylitis fracture | Hyperextension; even minor falls | High instability and cord injury risk |